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标题: 非核苷乙型肝炎病毒聚合酶抑制剂通过体外聚合酶延伸试验 [打印本页]

作者: StephenW    时间: 2020-3-23 12:58     标题: 非核苷乙型肝炎病毒聚合酶抑制剂通过体外聚合酶延伸试验

本帖最后由 StephenW 于 2020-3-23 13:00 编辑

Non-nucleoside hepatitis B virus polymerase inhibitors identified by an in vitro polymerase elongation assay

    Shogo Nakajima, Koichi Watashi, Kento Fukano, Senko Tsukuda, Kousho Wakae, Hideki Aizaki, Masamichi Muramatsu, Takaji Wakita & Tetsuya Toyoda

Journal of Gastroenterology volume 55, pages441–452(2020)Cite this article

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Abstract
Background

Hepatitis B virus (HBV) polymerase is the only virus-encoded enzyme essential for producing the HBV genome and is regarded as an attractive drug target. However, the difficulty of synthesizing and purifying recombinant HBV polymerase protein has hampered the development of new drugs targeting this enzyme, especially compounds unrelated to the nucleoside structure. We recently have developed a technique for the synthesis and purification of recombinant HBV polymerase containing the reverse transcriptase (RT) domain that carried DNA elongation activity in vitro.
Methods
We used the overproduced protein to establish an in vitro high-throughput screening system to identify compounds that inhibit the elongation activity of HBV polymerase.
ResultsWe screened 1120 compounds and identified a stilbene derivative, piceatannol, as a potential anti-HBV agent. Derivative analysis identified another stilbene derivative, PDM2, that was able to inhibit HBV replication with an IC50 of 14.4 ± 7.7 μM. An infection experiment suggested that the compounds inhibit the replication of HBV rather than the entry process, as expected. Surface plasmon resonance analysis demonstrated a specific interaction between PDM2 and the RT domain. Importantly, PDM2 showed similar inhibitory activity against the replication of both wild-type HBV and a lamivudine/entecavir-resistant HBV variant. Furthermore, PDM2 showed an additive effect in combination with clinically used nucleos(t)ide analogs.

Conclusions
We report the development of a screening system that is useful for identifying non-nucleos(t)ide RT inhibitors.


作者: StephenW    时间: 2020-3-23 12:58

本帖最后由 StephenW 于 2020-3-23 13:01 编辑

非核苷乙型肝炎病毒聚合酶抑制剂通过体外聚合酶延伸试验鉴定

     中岛正吾,渡边浩一,深野贤人,筑田信光,若草若江,荒木秀树,村松雅知,牧田孝司和丰田章男

胃肠病学杂志第55卷,第441–452页(2020年)

     184次访问

     1引文

     指标详细信息

抽象
背景

乙型肝炎病毒(HBV)聚合酶是唯一可产生HBV基因组的病毒编码酶,被视为有吸引力的药物靶标。 然而,合成和纯化重组HBV聚合酶蛋白的困难阻碍了针对该酶的新药的开发,特别是与核苷结构无关的化合物。 我们最近开发了一种合成和纯化重组HBV聚合酶的技术,该重组HBV聚合酶包含在体外具有DNA延伸活性的逆转录酶(RT)域。

方法

我们使用过量生产的蛋白质建立了体外高通量筛选系统,以鉴定抑制HBV聚合酶延伸活性的化合物。
结果

我们筛选了1120种化合物,并确定了一种二苯乙烯衍生物Piceatannol作为潜在的抗HBV药物。衍生分析确定了另一种二苯乙烯衍生物PDM2,其能够抑制HBV复制,IC50为14.4±±7.7μM。感染实验表明,这些化合物抑制了HBV的复制,而不是如预期的那样进入过程。表面等离子体共振分析表明PDM2和RT域之间的特定相互作用。重要的是,PDM2对野生型HBV和耐拉米夫定/恩替卡韦的HBV变异体的复制均显示出相似的抑制活性。此外,PDM2与临床使用的核苷酸(t)化物类似物组合显示出加和作用。
结论

我们报告了筛查系统的发展,该系统可用于鉴定非核苷(t)ide RT抑制剂。





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