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标题: Dis-Assembly:功能性治愈挫折引发乙型肝炎专家裁员 [打印本页]

作者: StephenW    时间: 2022-7-22 09:19     标题: Dis-Assembly:功能性治愈挫折引发乙型肝炎专家裁员

Dis-Assembly:功能性治愈挫折引发乙型肝炎专家裁员
尼克·保罗·泰勒
2022年7月21日晚上11点14分
Assembly BiosciencesArbutus Biopharmahepatitis Blayoffs
裁员, 裁员, 退出, 削减, 员工, 员工, 裁员, 劳动力减少
Assembly Biosciences 正在瘦身,以将其现金跑道延长至 2024 年上半年。(No-Mad/iStock/Getty Images Plus)

对两项三重组合研究的中期审查破坏了 Assembly Biosciences 围绕 vebicorvir 建立功能性治愈方案的计划。承认核心抑制剂将达不到要求,这家生物技术公司正在转向早期阶段的候选人——并裁员 30% 以勉强维持剩余的现金。

一年前,Assembly 正在通过 2 期临床试验与两种乙型肝炎前景竞争,vebicorvir 和 ABI-H2158。 ABI-H2158 在 9 月达到缓冲液,当时肝毒性增加促使 Assembly 停止候选药物的开发。受挫后,vebicorvir(也称为 VBR)的工作继续与 Arbutus Biopharma 和 Antios Therapeutics 合作。

现在,在 5 月份退出 Antios 合作后,Assembly 已经停止了 vebicorvir 的开发,以应对低迷的数据。与 Arbutus 合作的试验将继续进行到主要终点,数据将于 2022 年下半年公布,但 Assembly 放弃了将 vebicorvir 与核苷(酸)类似物逆转录酶抑制剂( NrtIs )结合的想法。

“通过将 VBR 与 NrtI 疗法相结合,我们实现了比单独使用 NrtI 更快速和更深层次的病毒抑制,并且我们继续看到 VBR 具有良好的安全性和耐受性。不幸的是,根据我们目前研究的中期数据,我们不相信任何一种 VBR 三联疗法都可能对慢性 HBV 感染患者实现有意义的功能治愈率,”Assembly 首席执行官 John McHutchison 医学博士在一篇文章中说陈述。
有关的
在一次 HBV 失败后,Assembly Biosciences 因安全问题被迫放弃肝炎资产

这一结论导致 Assembly 立即停止了自己的 2 期试验,同时同意将 Arbutus 研究进行到底。在砍掉 vebicorvir 之后,Assembly 现在专注于 ABI-H3733 和 ABI-4334。 ABI-H3733 正在进行 1b 期临床试验。初始数据应在年底前提交。 ABI-4334 计划于今年进入 1a 期研究。

McHutchison 说,这些候选药物“明显更有效,并提供了对抗 cccDNA 的二级机制”,一种被认为是慢性乙型肝炎根源的 DNA 形式。根据 McHutchison 的说法,二级机制可能“对有限和治愈性至关重要治疗乙肝病毒。”

Assembly 正在缩减规模,以将其现金跑道延长至 2024 年上半年。这家生物技术公司计划与 30 名员工分道扬镳,几乎是目前员工人数的三分之一。首席医疗官 Luisa Stamm,医学博士,博士,是离开大会的人之一。开发运营高级副总裁 Michele Anderson 将于下月初担任首席开发官一职。
作者: StephenW    时间: 2022-7-22 09:19

Dis-Assembly: Functional cure setback triggers layoffs at hepatitis B specialist
By Nick Paul Taylor
Jul 21, 2022 11:14pm
Assembly BiosciencesArbutus Biopharmahepatitis Blayoffs
layoffs, layoff, exit, cut, employees, staff, downsize, workforce reduction
Assembly Biosciences is slimming down to extend its cash runway into the first half of 2024. (No-Mad/iStock/Getty Images Plus)

An interim review of two triple combination studies has torpedoed Assembly Biosciences’ plan to build a functional cure regimen around vebicorvir. Accepting that the core inhibitor will fall short, the biotech is pivoting to earlier-stage candidates—and laying off 30% of staff to eke out its remaining cash.

One year ago, Assembly was racing two hepatitis B prospects, vebicorvir and ABI-H2158, through phase 2 clinical trials. ABI-H2158 hit the buffers in September when increased liver toxicity drove Assembly to stop development of the candidate. Work on vebicorvir, also known as VBR, continued in collaboration with Arbutus Biopharma and Antios Therapeutics in the wake of the setback.

Now, having pulled out of the Antios collaboration in May, Assembly has halted vebicorvir development in response to lackluster data. The Arbutus-partnered trial will continue to the primary endpoint, with data due in the second half of 2022, but Assembly is giving up on the idea of combining vebicorvir with nucleos(t)ide analogue reverse transcriptase inhibitors (NrtIs).

“By combining VBR with NrtI therapy, we achieved a more rapid and a deeper level of viral suppression than with NrtI alone and we have continued to see a favorable safety and tolerability profile for VBR. Unfortunately, we do not believe, based on the interim data from our current studies, that either VBR triple combination is likely to achieve a meaningful rate of functional cure for patients with chronic HBV infection,” Assembly CEO John McHutchison, M.D., said in a statement.
Related
After one HBV flop, Assembly Biosciences forced to drop a hepatitis asset amid safety woes

The conclusion led Assembly to immediately stop its own phase 2 trial while agreeing to see the Arbutus study through to the end. Having axed vebicorvir, Assembly is now focused on ABI-H3733 and ABI-4334. ABI-H3733 is in a phase 1b clinical trial. Initial data are due by the end of the year. ABI-4334 is scheduled to move into a phase 1a study this year.

McHutchison said the candidates “are significantly more potent and provide the secondary mechanism against cccDNA,” a form of DNA that is thought to be at the root of chronic hepatitis B. According to McHutchison, the secondary mechanism may be “critical to finite and curative treatments for HBV.”

Assembly is slimming down to extend its cash runway out into the first half of 2024. The biotech plans to part company with 30 employees, almost one-third of its current headcount. Chief Medical Officer Luisa Stamm, M.D., Ph.D., is among the people leaving Assembly. Michele Anderson, senior vice president of development operations, will take up the post of chief development officer at the start of next month.
作者: tim889    时间: 2022-7-22 09:55

想纯通过Core Inhibitor来耗竭cccDNA 恐怕本身也走不通
作者: tim889    时间: 2022-7-22 09:55

而且 之前的 Core Inhibitor 都容易产生耐药
作者: sky8989    时间: 2022-7-22 19:20

古老的病毒,世纪难题。如果能耗竭cccDNA那就彻底治愈了。现在连功能性治愈都是遥不可及,最期待的gsk与vir也不给力,可以看出目前的这些二期药物全部没戏了。哎,心灰意冷
作者: newchinabok    时间: 2022-7-22 19:32

新药搞不出明堂,早对新药不指望了




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